首页|动脉瘤性蛛网膜下腔出血后全脑水肿的危险因素、发病机制及诊疗研究进展

动脉瘤性蛛网膜下腔出血后全脑水肿的危险因素、发病机制及诊疗研究进展

Research progress in risk factors,pathogenesis,and treatment of global cerebral edema after aneurysmal subarachnoid hemorrhage

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动脉瘤性蛛网膜下腔出血(aSAH)后易并发全脑水肿,导致患者病情加重.因此,明确aSAH后全脑水肿(GCE)的危险因素、发病机制、诊断方法及治疗进展等,对降低GCE发生率,改善患者的预后具有重要意义.本文主要对aSAH后GCE的危险因素、发病机制及治疗进展等方面做一综述,以便对该类患者提前进行预防干预,改善患者的预后.
Cerebral edema is easily complicated after aneurysmal subarachnoid hemorrhage(aSAH),which leads to the aggravation of the patient's condition.Therefore,to clarify the risk factors,pathogenesis,diagnostic methods and treatment progress of global cerebral edema(GCE)after aSAH is of great significance for reducing the incidence of GCE and improving the prognosis of patients.This article mainly reviews the risk factors,pathogenesis,and treatment progress of GCE after aSAH,in order to provide early preventive intervention and improve the prognosis of such patients.

aneurysmal subarachnoid hemorrhageglobal cerebral edemarisk factorspathogenesistreatment progress

曾静、曾春、唐爽、向城卫、陈星宇

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563000 遵义,遵义医科大学

四川省遂宁市中心医院神经外科

动脉瘤性蛛网膜下腔出血 全脑水肿 危险因素 发病机制 治疗进展

四川省医学(青年创新)科研课题立项项目

Q22014

2024

临床神经外科杂志
南京医科大学附属脑科医院

临床神经外科杂志

CSTPCD
影响因子:1.019
ISSN:1672-7770
年,卷(期):2024.21(4)
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