世界中医药2024,Vol.19Issue(11) :1644-1648.DOI:10.3969/j.issn.1673-7202.2024.11.017

"血瘀证"视角下2型糖尿病合并泛血管疾病的关系

Relationship between Type 2 Diabetes Mellitus Complicated with Panvascular Disease from Perspective of"Blood Stasis Syndrome"

张仕衡 苏嘉楠 杨宇峰 石岩
世界中医药2024,Vol.19Issue(11) :1644-1648.DOI:10.3969/j.issn.1673-7202.2024.11.017

"血瘀证"视角下2型糖尿病合并泛血管疾病的关系

Relationship between Type 2 Diabetes Mellitus Complicated with Panvascular Disease from Perspective of"Blood Stasis Syndrome"

张仕衡 1苏嘉楠 2杨宇峰 3石岩2
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作者信息

  • 1. 辽宁中医药大学,沈阳,110847;中医脏象理论及应用教育部重点实验室,沈阳,110847;辽宁中医药大学中医药创新工程技术中心,沈阳,110847
  • 2. 辽宁中医药大学,沈阳,110847
  • 3. 辽宁中医药大学,沈阳,110847;辽宁中医药大学中医学院,沈阳,110847
  • 折叠

摘要

泛血管疾病是2型糖尿病(T2DM)的常见合并疾病,也是T2DM患者致残致死的重要原因,因而预防T2DM合并泛血管疾病的病情进展是临床关注的重点.现代理论研究多将T2DM归属于"消渴"范畴,通过整理T2DM合并泛血管疾病的病因病机和机制研究,分析消渴心病、消渴肾病、消渴目病的因机内核,确定其核心为血瘀证,瘀血又作为其病理产物贯穿始终.

Abstract

Panvascular disease is a common complication of type 2 diabetes mellitus(T2DM)and an important cause of disability and death in patients with T2DM.Therefore,delaying the progression of T2DM complicated with panvascular disease is the focus of clinical attention.Modern theoretical research mostly attributes T2DM to Xiaoke diabetes.This paper analyzed the etiology,patho-genesis,and mechanism of T2DM complicated with panvascular disease and investigated the pathogenesis of Xiaoke diabetes heart disease,Xiaoke diabetes kidney disease,and Xiaoke diabetes eye disease.The core of the disease was determined to be blood stasis syndrome,and blood stasis is its pathological product throughout the disease development.

关键词

血瘀证/2型糖尿病/泛血管疾病/并发症/病因病机/中医理论/病理机制/中医药

Key words

Blood stasis syndrome/Type 2 diabetes mellitus/Panvascular disease/Complication/Etiology and pathogenesis/Traditional Chinese medicine theory/Pathomechanism/Traditional Chinese medicine

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基金项目

国家重点基础研究发展计划(973计划)项目(2013CB532004)

国家中医药领军人才支持计划-岐黄学者项目([2018]12)

辽宁省"兴辽英才计划"青年拔尖人才项目(XLYC2007121)

辽宁省人社厅百千万人才资助项目(20200512)

辽宁省糖尿病中医病证结合重点实验室(2019JH6/10100009)

出版年

2024
世界中医药
世界中医药学会联合会

世界中医药

CSTPCDCHSSCD北大核心
影响因子:1.266
ISSN:1673-7202
参考文献量11
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