首页|围产期母畜低血钙症的钙代谢机制及对相关代谢疾病的影响

围产期母畜低血钙症的钙代谢机制及对相关代谢疾病的影响

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围产期母畜由于体内蛋白质、矿物质和能量代谢的急剧变化,容易导致钙的流失和平衡失调,进而引发母畜围产期低血钙症.临床和亚临床低血钙症不仅能够影响母畜的生产性能,还会影响围产期机体的营养代谢,进一步诱发母畜在围产期出现其他生理变化和疾病,如酮病、皱胃移位、难产、乳腺炎及子宫疾病等.因此,全面深入地研究围产期母畜低血钙症的钙代谢机制及其对相关代谢疾病的影响,对于保障围产期母畜的健康和提高生产性能具有重要意义.文章详述了围产期母畜低血钙症的病因和现状、钙代谢机制、综合防治措施以及与其他疾病之间的关联,为降低围产期母畜低血钙症发病率以及改善饲养管理提供参考.
Calcium metabolism mechanism and its impact on related metabolic disorders in periparturient hypocalcemia of periparturient females
During the perinatal period,changes in the metabolism of proteins,minerals,and energy in periparturient dams can lead to calcium loss and imbalance,subsequently causing hypocalcemia in periparturient dams.Clinical and subclinical hypocalcemia not only affects the reproductive performance of dams but also influences the nutritional metabolism of the periparturient organism,further triggering other physiological changes and diseases in periparturient dams,such as ketosis,displaced abomasum,dystocia,mastitis,and uterine diseases.Therefore,a comprehensive and in-depth study of the calcium metabolism mechanism during periparturient hypocalcemia and its impact on related metabolic diseases is of significant importance for ensuring the health of periparturient dams and improving their reproductive performance.This article elaborates on the etiology and current status of periparturient hypocalcemia in dams,calcium metabolism mechanisms,comprehensive preventive and control measures,as well as the correlations with other diseases.It serves as a reference for reducing the incidence of periparturient hypocalcemia in dams and improving feeding management.

periparturient femaleshypocalcemiacalcium metabolismmetabolic disorders

义拉勒特、金鹿、张春华、萨初拉、李胜利、张崇志、刘威、孙海洲

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内蒙古农业大学动物科学学院,内蒙古呼和浩特 010018

内蒙古自治区农牧业科学院内蒙古自治区草食动物营养科学重点实验室农业农村部草食家畜健康养殖与畜产品品质调控重点室(部省共建),内蒙古呼和浩特 010031

围产期母畜 低血钙症 钙代谢 代谢疾病

国家绒毛用羊产业技术体系"科技兴蒙"行动重点专项内蒙古自然科学基金项目内蒙古自治区农业科学院青年基金项目农业农村部草食家畜健康养殖与畜产品品质调控重点室(部省共建)项目

CARS-39-11KJXM-EEDS-20200022021LHMS030132022CXJJM14

2024

饲料研究
北京市营养源研究所

饲料研究

CSTPCD北大核心
影响因子:0.391
ISSN:1002-2813
年,卷(期):2024.47(3)
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