AhR mediates a broad range of biological responses to environmental chemicals, including suppression of immune functions. In this issue of Blood, Lawrence and colleagues demonstrate that AhR is responsible for the anti-inflammatory activity of new antiallergic drug candidates, likely by blocking dendritic-cell function to generate proinflammatory T-helper cells.
receptorXenobioticsInflammation异生物质炎症
Ma Q
展开 >
National Institute for Occupational Safety and Health.
NSTL
