Stanford B型主动脉夹层腔内修复术后主动脉扩张性病变的研究进展
Research progress in aortic dilatation lesions occurring after endovascular repair of Stanford type B aortic dissection
李天祎1
作者信息
- 1. 315010 浙江宁波 宁波大学附属第一医院影像科
- 折叠
摘要
胸主动脉腔内修复术(thoracic endovascular aortic repair,TEVAR)目前是Stanford B型主动脉夹层(type B aortic dissection,TBAD)的主要治疗方法,当下治疗原则以封堵近端破口为主,而旷置远端破口导致假腔血栓化受阻;主动脉形态及支架远端弹性应力变化,导致管腔局部点压力扩大,形成新发破口或再发夹层导致假腔瘤样扩张,甚至急性破裂致死.术后应通过定期CTA随访,严密监测支架远端管径变化、残留破口直径及假腔血栓化程度.本文通过总结国内外文献,对TEVAR后主动脉扩张性病变的发病原因、机制及处理方法等进行归纳并作综述.
Abstract
At present,thoracic endovascular aortic repair(TEVAR)is the main therapeutic method for Stanford type B aortic dissection(TBAD).The current treatment principle is mainly to occlude the proximal rupture,while the distal rupture is left open,causing the false lumen unable to become thrombosis.Changes in aortic morphology and elastic stress at the distal end of the stent will lead to increased pressure at the local point of the lumen and produce a new rupture or recurrent dissection,thus,causing tumor-like expansion of the false lumen,or even leading to acute rupture and death.After operation,regular CTA follow-up is required so as to closely monitor the changes in the distal stent diameter,the residual rupture diameter and the degree of thrombosis within the false lumen.By reviewing the domestic and foreign literature,this article summarizes the causes,mechanisms and treatment of aortic dilatation lesions occurring after TEVAR for TBAD.(J Intervent Radiol,2024,33:443-448)
关键词
胸主动脉腔内修复术/主动脉夹层/支架移植物远端新发破口Key words
thoracic endovascular aortic repair/aortic dissection/distal stent graft-induced new entry引用本文复制引用
出版年
2024
NETL
NSTL
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