摘要
在畜禽繁殖体系中,种公鸡发挥着关键作用,公鸡精子活力是评价种公鸡繁殖性能的重要指标.线粒体是维持公鸡精子正常运动的关键细胞器之一,主要通过氧化磷酸化产生ATP为精子的运动和受精过程提供所需的动力.当线粒体出现结构损伤、功能障碍、基因突变、mtDNA损伤时,会导致精子活力降低.因此,研究公鸡精子线粒体对精子活力的调控机制对于提升精液品质和优化家禽繁殖性能具有重要的理论和实践价值.文章综述了线粒体结构、线粒体基因和mtDNA拷贝数对精子活力的调控机制,以期为提高精子活力和优化家禽繁殖性能提供理论基础.
Abstract
Breeder roosters are a crucial part in the livestock and poultry breeding system,and the sperm motility of roosters is one of the most important indicator for evaluating the reproductive performance of breeder roosters.The mitochondria,being one of the organelles crucial for maintaining sperm motility in spermatozoa,play a pivotal role in generating ATP through oxidative phosphorylation,and ATP is indispensable for sperm motility and fertilization.When mitochondria undergo structural damage,dysfunction,genetic mutations,or mtDNA damage,it could lead to a decrease sperm motility.Researching the mechanism by which sperm mitochondria regulate sperm motility in roosters'spermatozoa holds significant theoretical and practical value,as it might enhance semen quality and poultry reproductive performance.This paper provided a comprehensive review of the regulatory mechanisms that govern mitochondrial structure,mitochondrial genes,and mtDNA copy numbers on sperm motility,and aimed to establish a theoretical foundation for enhancing sperm motility and optimizing poultry reproductive performance.
NETL
NSTL
万方数据